Meet Inspiring Speakers and Experts at our 3000+ Global Conference Series Events with over 1000+ Conferences, 1000+ Symposiums
and 1000+ Workshops on Medical, Pharma, Engineering, Science, Technology and Business.

Explore and learn more about Conference Series : World's leading Event Organizer

Back

International Conference on Neurological disorder and Neuroimmunology

Montreal, Canada

Zeinab Norouzi

Lorestan University of Medical Sciences, Iran

Title: Reporting a patient with Chronic Cardiac tamponade in a life threatening disease (Tuberculosis Pericarditis)

Biography

Biography: Zeinab Norouzi

Abstract

Cardiac tamponade happens when the collection of fluid in pericardial space is more quickly than the expansion of the pericardial sac to incorporate the excess fluid. It can cause a high pressure in pericardial sac and prevents the effective heart contraction. In acute tamponade, a small amount of fluid can cause problem and even death for the patient, but in chronic tamponade, the pericardial sac can stretch to hold more than even 1000 mL, without significant symptoms in patient. This form of tamponade happens in tuberculosis pericarditis. We report a patient with chronic tamponade due to tuberculosis
pericarditis. A 13 year old female, presented to the clinic with hematuria, dysuria, fatigue and peripheral edema. She was completely comfortable and the vital signs were stable. A computerized tomography (CT) scan of abdomen and pelvis was ordered that revealed moderate ascites, right-sided pleural effusion, and massive pericardial effusion. She was admitted to the hospital. The positive signs in complete physical exam was rising of JVP, decreased the respiratory sounds in right hemithorax,paradoxical pulse and 1+ edema in the lower limbs. Also, the heart sounds were muffled. The initial ECG showed tachycardia and low voltage of QRS complex. Echocardiographic findings were 3.6 cm pericardial effusion and the collapse of right ventricle in diastole. Pericardio synthesis and pericardial window were recommended and 2500 mL of bloody fluid was drained. Pericardial effusion analysis showed lymphocyte dominancy and a high level of ADA (80 U/L). The work up for TB was negative. According to the symptoms, pericardial and pleural effusion and high level of ADA and living in endemic area for TB, empiric therapy was initiated and the response of the patient was excellent without any complications in two months follow up.